What is the diathesis-stress hypothesis? How does it explain the development of schizophrenia?

Short Answer

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The diathesis-stress hypothesis suggests that mental disorders like schizophrenia are a result of a biological or genetic vulnerability (diathesis) that, when combined with stressful life events or situations (stress), leads to the development of the disorder. In other words, both a predisposition and a triggering event are necessary for the onset of schizophrenia.

Step by step solution

01

Understanding the Diathesis-Stress Hypothesis

The diathesis-stress hypothesis is a psychological theory which tries to explain why we develop mental disorders. The word 'diathesis' refers to a physical or biological vulnerability or predisposition to a condition. 'Stress' refers to life stressors or significant life events that might trigger the condition in someone with the inherent vulnerability.
02

Linking the Hypothesis to Schizophrenia

Applying this hypothesis to schizophrenia, the diathesis or predisposition could be a biological vulnerability such as genetics or abnormalities in the brain. The stressor could be a traumatic event, high levels of stress, or even substance abuse. For an individual with the biological predisposition (diathesis), experiencing these high levels of stress could trigger the onset of schizophrenia.
03

Elaborating on the Development of Schizophrenia

Not everyone with a biological predisposition develops schizophrenia; it typically only develops once triggered by significant life stressors. Thus, an individual might carry a genetic vulnerability for schizophrenia, but unless they encounter certain environmental stressors, they may not develop the condition. This interplay between genetic predisposition and environmental factors is at the crux of the diathesis-stress hypothesis for the development of schizophrenia.

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